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The success of AutoCAD Activation Code led to the release of AutoCAD LT (a smaller, portable version) in 1985 and later AutoCAD Graphics in 1992, AutoCAD R14 in 1993, AutoCAD LT for Windows in 1996, AutoCAD Map 3D in 1998, AutoCAD Map 3D for ArcGIS in 2005, and AutoCAD R18 in 2010.

AutoCAD was originally released for MS-DOS operating systems and was developed using the Object Linking and Embedding (OLE) technology of Microsoft. Since version 16, AutoCAD has supported macOS. AutoCAD 2018, launched in August 2018, supports Ubuntu Linux, Fedora, and Red Hat Linux operating systems.

Features

AutoCAD is capable of drafting architectural and mechanical designs, and creating construction documents, including construction plans and blueprints. The software was designed to enable architects and engineers to create and edit complex designs such as 3D models, which can then be exported to other applications.

Interface

The design interface of AutoCAD is the principle interface, the rest of the design process is handled through AutoCAD commands and the AutoCAD tools. AutoCAD commands are located in the top right corner of the window, and most of the commands operate on the selected object. The commands can be used from any tool. The layout of the user interface is similar to that of AutoCAD’s predecessor, Solidworks.

Program Architecture

The AutoCAD program architecture and design is composed of four layers;

The Application Layer,

The Tools Layer,

The Raster Graphics Layer, and

The Application Database Layer.

AutoCAD is made up of 1,600 functions, and a command button can be accessed by entering an argument at the command line. The command buttons themselves are a part of the Application Layer. If the user wishes to make changes to a command or the command menu, this is performed by using functions in the Tools Layer.

The Application Database Layer contains information about the project being worked on and the project’s history, such as the number of commands and toolbars that have been used. If a command cannot be found, then the application raises an error that appears in the Status Bar, and this information is displayed in the tooltip box.

The Application Layer comprises the AutoCAD commands, a command manager, and the command line. It is here that the

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HTML/JS apps that use internet protocols to control the software through the web browser.

On 25 September 2019, Autodesk acquired Onshape, an augmented reality CAD application.

References

Further reading

(Published by Autodesk)

(Published by Autodesk)

External links

Blog
User forums
AutoCAD product reviews and video

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Category:Software companies of the United StatesPIM1 knockdown selectively impairs the induction of CD24-EpCAM double-positive cell surface markers during T cell activation.
We established a human embryonic kidney cell line, 293T-PIM1, transfected with a small interfering RNA (siRNA) for PIM1 gene expression, and examined its effects on the activation of T cells. PIM1 siRNA specifically inhibited PIM1 expression in 293T-PIM1. PIM1 siRNA-transfected cells showed growth arrest at the G1/S phase transition, accompanied by decreased CD3 and CD28 expression levels. The CD3(-) CD56(+) population was also reduced by the PIM1 siRNA transfection. Flow cytometry analysis showed that the proportion of CD3(+) CD56(+) CD16(-) cells, expressing CD24-EpCAM double-positive markers, were greatly increased when PBMCs were activated by anti-CD3 and anti-CD28 antibodies, and the CD3(+) CD56(+) CD16(-) cells decreased in proportion by the PIM1 siRNA transfection. Our data suggest that PIM1 knockdown selectively impairs the induction of the CD24-EpCAM double-positive cell surface markers during T cell activation.Q:

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Identification of genome-specific up-regulated miRNAs in methanol-induced maize callus.
Methanol is an important in vitro micropropagation system for many crops. However, the molecular mechanism of methanol-induced callus induction and growth remains unknown. In the present study, a total of 1060 differentially expressed miRNAs, including 6 up-regulated and 4 down-regulated miRNAs, were identified between methanol-treated calli and control calli using a high-throughput sequencing technique. The identification of differentially expressed miRNAs was performed using a statistical algorithm and the bioinformatics software package Targetfinder. The results indicated that members of the miR166 and miR395 families were most significantly up-regulated in response to methanol. The two major histidine-containing phoshatase (HPT) transcripts were post-transcriptionally regulated by miR166, and miR395-targeted miRNAs have been shown to be involved in phosphate homeostasis. The expression of genes that were predicted to be target genes of the two miRNAs was analyzed to confirm the post-transcriptional regulation of miR166 and miR395 in methanol-induced callus. Gene Ontology (GO) analysis of the differentially expressed genes (DEGs) between the two calli was performed to predict their biological functions. The results revealed that the miRNAs are key players in methanol-induced maize callus. Our findings might be useful for understanding the molecular mechanism of in vitro micropropagation in maize.Pages

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Expand the full ribbon to see symbols and functions that match your most used commands, such as Undo, Save and Save As. Save time by diving into your AutoCAD settings to enable the right options for your workflow.

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