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“Till My Last Day” is a song written by John Anderson and Leslie Satcher, and recorded by American country music artist Dierks Bentley. It was released in January 2006 as the third single from his album If These Arms Could Whisper. The song became his third consecutive number one hit on the U.S. Billboard Hot Country Songs chart.
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The song describes the narrator’s wish that he could talk to his lover with his last breath, and tell her of all the love he feels for her.
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The music video was directed by Donovan Cook and premiered in early-2006.
Chart performance
Year-end charts
References
External links
Category:2005 singles
Category:2006 singles
Category:Dierks Bentley songs
Category:Billboard Hot Country Songs number-one singles
Category:Song recordings produced by Brett Beavers
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Mutations in TSC2 are responsible for tuberous sclerosis complex (TSC) in patients with mental retardation, seizures, and characteristic tumors. While the majority of TSC patients have mental retardation as the only symptom, there are cases of TSC with multiple renal cysts in addition to mental retardation. Interestingly, renal cysts and mental retardation frequently occur in patients with TSC2 mutations. Here we report a Japanese female with multiple renal cysts and very mild TSC-like manifestations who had three different missense mutations in TSC2. In addition to multiple renal cysts, she had two histologically proven pilocytic astrocytomas, but lacked the characteristic features of other TSC-related manifestations such as facial angiofibromas, rhabdomyomas in the heart, and hemangiomas. The mutations c.844G > A (p.Gly282Ser) in exon 8, c.1129C > T (p.Pro377Ser) in exon 9, and c
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Epigallocatechin-3-gallate (EGCG), a polyphenol antioxidant component of green tea, is thought to have chemopreventive properties. We examined the effects of EGCG and its stereoisomer, epigallocatechin-3-O-gallate (EGCG-O-gallate), on a murine colon carcinoma cell line and human colon adenocarcinoma cell lines. Cultured cell lines were treated with EGCG, EGCG-O-gallate, or dimethyl sulfoxide (DMSO) as a vehicle control for 24, 48, or 72 h. Cell proliferation was evaluated by MTT assay, mitochondrial dehydrogenase activity, and flow cytometry using ethidium bromide/propidium iodide staining. EGCG inhibited cell proliferation and clonogenic survival of the murine colon cancer cell line in a dose- and time-dependent manner (0-100 microM and 24-72 h), with minimum inhibitory concentration of approximately 10 microM at 72 h. The EGCG isomer was ineffective. EGCG was also cytotoxic to human colon adenocarcinoma cell lines Caco-2 and HT-29, with IC(50) values of 18.5 +/- 1.5 microM and 6.0 +/- 1.4 microM, respectively. A time- and concentration-dependent growth inhibitory effect on both cell lines was also observed with EGCG (0-100 microM). The isomer, however, was not cytotoxic. EGCG induced G(1) phase arrest in the human colon adenocarcinoma cell lines, whereas EGCG-O-gallate induced apoptosis. These effects were mitogen-responsive and did not require intact cell-cell interactions, indicating that EGCG may inhibit colon carcinoma cell proliferation by inducing G(1) phase cell cycle arrest. Because the isomer was not active, the increased activity of EGCG may be attributed to its stereochemistry., United States v. LaBonte, 520 U.S. 751, 117 S.Ct. 1673, 137 L.Ed.2d 1001 (1997). For example, the prisoner “must have received process
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